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The PTB Domain: The Name Doesn't Say It All.

Trends in endocrinology and metabolism: TEM

Margolis B.
PMID: 10461172
Trends Endocrinol Metab. 1999 Sep;10(7):262-267. doi: 10.1016/s1043-2760(99)00168-x.

The phosphotyrosine-binding domain is a recently described protein-protein interaction domain which, despite its name, is involved in both phosphotyrosine-dependent and -independent interactions. Proteins with this domain are involved in diverse cellular functions, ranging from receptor signaling to protein targeting.

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Margolis B. The PTB Domain: The Name Doesn't Say It All. Trends Endocrinol Metab. 1999;10(7):262-267doi: 10.1016/s1043-2760(99)00168-x.
Margolis, B. (1999). The PTB Domain: The Name Doesn't Say It All. Trends in endocrinology and metabolism: TEM, 10(7), 262-267. https://doi.org/10.1016/s1043-2760(99)00168-x
Margolis. "The PTB Domain: The Name Doesn't Say It All." Trends in endocrinology and metabolism: TEM vol. 10,7 (1999): 262-267. doi: https://doi.org/10.1016/s1043-2760(99)00168-x
Margolis B. The PTB Domain: The Name Doesn't Say It All. Trends Endocrinol Metab. 1999 Sep;10(7):262-267. doi: 10.1016/s1043-2760(99)00168-x. PMID: 10461172.
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Mechanisms of Glucocorticoid-receptor-mediated Repression of Gene Expression.

Trends in endocrinology and metabolism: TEM

Webster JC, Cidlowski JA.
PMID: 10542396
Trends Endocrinol Metab. 1999 Dec;10(10):396-402. doi: 10.1016/s1043-2760(99)00186-1.

It is hoped that this review will give the reader a taste of some of the mechanisms used by the glucocorticoid receptor to repress gene function. These mechanisms include direct binding to DNA, antagonism of other transcription factor families...

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Webster JC, Cidlowski JA. Mechanisms of Glucocorticoid-receptor-mediated Repression of Gene Expression. Trends Endocrinol Metab. 1999;10(10):396-402doi: 10.1016/s1043-2760(99)00186-1.
Webster, J. C., & Cidlowski, J. A. (1999). Mechanisms of Glucocorticoid-receptor-mediated Repression of Gene Expression. Trends in endocrinology and metabolism: TEM, 10(10), 396-402. https://doi.org/10.1016/s1043-2760(99)00186-1
Webster, and Cidlowski. "Mechanisms of Glucocorticoid-receptor-mediated Repression of Gene Expression." Trends in endocrinology and metabolism: TEM vol. 10,10 (1999): 396-402. doi: https://doi.org/10.1016/s1043-2760(99)00186-1
Webster JC, Cidlowski JA. Mechanisms of Glucocorticoid-receptor-mediated Repression of Gene Expression. Trends Endocrinol Metab. 1999 Dec;10(10):396-402. doi: 10.1016/s1043-2760(99)00186-1. PMID: 10542396.
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Real-time Analysis of Glucose Metabolism by Microscopy.

Trends in endocrinology and metabolism: TEM

Piston DW, Knobel SM.
PMID: 10542399
Trends Endocrinol Metab. 1999 Dec;10(10):413-417. doi: 10.1016/s1043-2760(99)00204-0.

Glucose metabolism has traditionally been assayed via biochemical means. Fluorescence monitoring of NAD(P)H levels has provided a non-invasive method to assay glucose metabolism in cells and tissues. However, these measurements have traditionally been of low resolution (no subcellular information)...

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Piston DW, Knobel SM. Real-time Analysis of Glucose Metabolism by Microscopy. Trends Endocrinol Metab. 1999;10(10):413-417doi: 10.1016/s1043-2760(99)00204-0.
Piston, D. W., & Knobel, S. M. (1999). Real-time Analysis of Glucose Metabolism by Microscopy. Trends in endocrinology and metabolism: TEM, 10(10), 413-417. https://doi.org/10.1016/s1043-2760(99)00204-0
Piston, and Knobel. "Real-time Analysis of Glucose Metabolism by Microscopy." Trends in endocrinology and metabolism: TEM vol. 10,10 (1999): 413-417. doi: https://doi.org/10.1016/s1043-2760(99)00204-0
Piston DW, Knobel SM. Real-time Analysis of Glucose Metabolism by Microscopy. Trends Endocrinol Metab. 1999 Dec;10(10):413-417. doi: 10.1016/s1043-2760(99)00204-0. PMID: 10542399.
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ErratumGenetic Defects in Postsqualene Cholesterol Biosynthesis.

Trends in endocrinology and metabolism: TEM

Moebius FF, Fitzky BU, Glossmann H.
PMID: 10754537
Trends Endocrinol Metab. 2000 May;11(4):150. doi: 10.1016/s1043-2760(00)00261-7.

No abstract available.

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Moebius FF, Fitzky BU, Glossmann H. ErratumGenetic Defects in Postsqualene Cholesterol Biosynthesis. Trends Endocrinol Metab. 2000;11(4):150doi: 10.1016/s1043-2760(00)00261-7.
Moebius, F. F., Fitzky, B. U., & Glossmann, H. (2000). ErratumGenetic Defects in Postsqualene Cholesterol Biosynthesis. Trends in endocrinology and metabolism: TEM, 11(4), 150. https://doi.org/10.1016/s1043-2760(00)00261-7
Moebius, et al. "ErratumGenetic Defects in Postsqualene Cholesterol Biosynthesis." Trends in endocrinology and metabolism: TEM vol. 11,4 (2000): 150. doi: https://doi.org/10.1016/s1043-2760(00)00261-7
Moebius FF, Fitzky BU, Glossmann H. ErratumGenetic Defects in Postsqualene Cholesterol Biosynthesis. Trends Endocrinol Metab. 2000 May;11(4):150. doi: 10.1016/s1043-2760(00)00261-7. PMID: 10754537.
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Complex Complexes: Report of an NIDDK Workshop on Coactivators and Corepressors, Bethesda, USA, 15-16 December 1998.

Trends in endocrinology and metabolism: TEM

Margolis R.
PMID: 18403278
Trends Endocrinol Metab. 1999 Sep;10(7):286-9. doi: 10.1016/s1043-2760(99)00165-4.

Exciting new observations about receptor-interacting and nuclear accessory proteins and their ability to regulate gene expression have provided insight into hormonal regulation of gene expression. The goal of a recent NIDDK workshop was to discuss how diverse signaling pathways...

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Margolis R. Complex Complexes: Report of an NIDDK Workshop on Coactivators and Corepressors, Bethesda, USA, 15-16 December 1998. Trends Endocrinol Metab. 1999;10(7):286-9doi: 10.1016/s1043-2760(99)00165-4.
Margolis, R. (1999). Complex Complexes: Report of an NIDDK Workshop on Coactivators and Corepressors, Bethesda, USA, 15-16 December 1998. Trends in endocrinology and metabolism: TEM, 10(7), 286-9. https://doi.org/10.1016/s1043-2760(99)00165-4
Margolis, R. "Complex Complexes: Report of an NIDDK Workshop on Coactivators and Corepressors, Bethesda, USA, 15-16 December 1998." Trends in endocrinology and metabolism: TEM vol. 10,7 (1999): 286-9. doi: https://doi.org/10.1016/s1043-2760(99)00165-4
Margolis R. Complex Complexes: Report of an NIDDK Workshop on Coactivators and Corepressors, Bethesda, USA, 15-16 December 1998. Trends Endocrinol Metab. 1999 Sep;10(7):286-9. doi: 10.1016/s1043-2760(99)00165-4. PMID: 18403278.
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Growth hormone-releasing peptide and its analogues Novel stimuli to growth hormone release.

Trends in endocrinology and metabolism: TEM

Korbonits M, Grossman AB.
PMID: 18406682
Trends Endocrinol Metab. 1995 Mar;6(2):43-9. doi: 10.1016/1043-2760(94)00204-h.

GHRPs are oligopeptides with GH-releasing effects in humans when given by either parenteral or oral routes; in addition, nonpeptide pharmacologic analogues have recently been synthesized. Although the exact mechanism of action of these agents has not been fully established,...

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Korbonits M, Grossman AB. Growth hormone-releasing peptide and its analogues Novel stimuli to growth hormone release. Trends Endocrinol Metab. 1995;6(2):43-9doi: 10.1016/1043-2760(94)00204-h.
Korbonits, M., & Grossman, A. B. (1995). Growth hormone-releasing peptide and its analogues Novel stimuli to growth hormone release. Trends in endocrinology and metabolism: TEM, 6(2), 43-9. https://doi.org/10.1016/1043-2760(94)00204-h
Korbonits, M, and Grossman, A B. "Growth hormone-releasing peptide and its analogues Novel stimuli to growth hormone release." Trends in endocrinology and metabolism: TEM vol. 6,2 (1995): 43-9. doi: https://doi.org/10.1016/1043-2760(94)00204-h
Korbonits M, Grossman AB. Growth hormone-releasing peptide and its analogues Novel stimuli to growth hormone release. Trends Endocrinol Metab. 1995 Mar;6(2):43-9. doi: 10.1016/1043-2760(94)00204-h. PMID: 18406682.
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Unraveling the human progesterone receptor signal transduction pathway Insights into antiprogestin action.

Trends in endocrinology and metabolism: TEM

McDonnell DP.
PMID: 18406695
Trends Endocrinol Metab. 1995 May-Jun;6(4):133-8. doi: 10.1016/1043-2760(95)00065-p.

The steroid hormone progesterone is a key modulator of the cellular processes that are required for the development and maintenance of reproductive function. Produced primarily by ovarian granulosa cells, it mediates its biological activity throughout the body by interacting...

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McDonnell DP. Unraveling the human progesterone receptor signal transduction pathway Insights into antiprogestin action. Trends Endocrinol Metab. 1995;6(4):133-8doi: 10.1016/1043-2760(95)00065-p.
McDonnell, D. P. (1995). Unraveling the human progesterone receptor signal transduction pathway Insights into antiprogestin action. Trends in endocrinology and metabolism: TEM, 6(4), 133-8. https://doi.org/10.1016/1043-2760(95)00065-p
McDonnell, D P. "Unraveling the human progesterone receptor signal transduction pathway Insights into antiprogestin action." Trends in endocrinology and metabolism: TEM vol. 6,4 (1995): 133-8. doi: https://doi.org/10.1016/1043-2760(95)00065-p
McDonnell DP. Unraveling the human progesterone receptor signal transduction pathway Insights into antiprogestin action. Trends Endocrinol Metab. 1995 May-Jun;6(4):133-8. doi: 10.1016/1043-2760(95)00065-p. PMID: 18406695.
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Nongenomic actions of progesterone on human spermatozoa.

Trends in endocrinology and metabolism: TEM

Baldi E, Krausz C, Forti G.
PMID: 18406702
Trends Endocrinol Metab. 1995 Aug;6(6):198-205. doi: 10.1016/1043-2760(95)00083-t.

Progesterone's (P) stimulatory actions on human spermatozoa have been known for many years. P indeed appears to be the main sperm stimulator present in women's biological fluids, particularly the follicular fluid. The nongenomic nature of the biological effects of...

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Baldi E, Krausz C, Forti G. Nongenomic actions of progesterone on human spermatozoa. Trends Endocrinol Metab. 1995;6(6):198-205doi: 10.1016/1043-2760(95)00083-t.
Baldi, E., Krausz, C., & Forti, G. (1995). Nongenomic actions of progesterone on human spermatozoa. Trends in endocrinology and metabolism: TEM, 6(6), 198-205. https://doi.org/10.1016/1043-2760(95)00083-t
Baldi, E, et al. "Nongenomic actions of progesterone on human spermatozoa." Trends in endocrinology and metabolism: TEM vol. 6,6 (1995): 198-205. doi: https://doi.org/10.1016/1043-2760(95)00083-t
Baldi E, Krausz C, Forti G. Nongenomic actions of progesterone on human spermatozoa. Trends Endocrinol Metab. 1995 Aug;6(6):198-205. doi: 10.1016/1043-2760(95)00083-t. PMID: 18406702.
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New frontiers in insulin receptor substrate signaling.

Trends in endocrinology and metabolism: TEM

Myers MG, White MF.
PMID: 18406704
Trends Endocrinol Metab. 1995 Aug;6(6):209-15. doi: 10.1016/1043-2760(95)00092-v.

Although most tyrosine kinase growth factor receptors directly bind Src homology 2 (SH2) proteins, the insulin receptor, and a select group of other hormone receptors-including an emerging group of cytokine receptors-phosphorylate intracellular insulin receptor substrate (IRS) proteins, which subsequently...

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Myers MG, White MF. New frontiers in insulin receptor substrate signaling. Trends Endocrinol Metab. 1995;6(6):209-15doi: 10.1016/1043-2760(95)00092-v.
Myers, M. G., & White, M. F. (1995). New frontiers in insulin receptor substrate signaling. Trends in endocrinology and metabolism: TEM, 6(6), 209-15. https://doi.org/10.1016/1043-2760(95)00092-v
Myers, M G, and White, M F. "New frontiers in insulin receptor substrate signaling." Trends in endocrinology and metabolism: TEM vol. 6,6 (1995): 209-15. doi: https://doi.org/10.1016/1043-2760(95)00092-v
Myers MG, White MF. New frontiers in insulin receptor substrate signaling. Trends Endocrinol Metab. 1995 Aug;6(6):209-15. doi: 10.1016/1043-2760(95)00092-v. PMID: 18406704.
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Apparent mineralocorticoid excess, 11beta hydroxysteroid dehydrogenase and aldosterone action Closing one loop, opening another.

Trends in endocrinology and metabolism: TEM

Funder JW.
PMID: 18406708
Trends Endocrinol Metab. 1995 Sep;6(7):248-51. doi: 10.1016/1043-2760(95)95219-u.

The recent cloning of the human enzyme 11beta hydroxysteroid dehydrogenase type 2 (11betaHSD2), and the demonstration of point mutations or deletions in both familial and apparently sporadic cases of apparent mineralocorticoid excess (AME), underlines the importance of this enzyme...

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Funder JW. Apparent mineralocorticoid excess, 11beta hydroxysteroid dehydrogenase and aldosterone action Closing one loop, opening another. Trends Endocrinol Metab. 1995;6(7):248-51doi: 10.1016/1043-2760(95)95219-u.
Funder, J. W. (1995). Apparent mineralocorticoid excess, 11beta hydroxysteroid dehydrogenase and aldosterone action Closing one loop, opening another. Trends in endocrinology and metabolism: TEM, 6(7), 248-51. https://doi.org/10.1016/1043-2760(95)95219-u
Funder, J W. "Apparent mineralocorticoid excess, 11beta hydroxysteroid dehydrogenase and aldosterone action Closing one loop, opening another." Trends in endocrinology and metabolism: TEM vol. 6,7 (1995): 248-51. doi: https://doi.org/10.1016/1043-2760(95)95219-u
Funder JW. Apparent mineralocorticoid excess, 11beta hydroxysteroid dehydrogenase and aldosterone action Closing one loop, opening another. Trends Endocrinol Metab. 1995 Sep;6(7):248-51. doi: 10.1016/1043-2760(95)95219-u. PMID: 18406708.
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Mechanism(s) of early glucocorticoid inhibition of adrenocorticotropin secretion from anterior pituitary corticotropes.

Trends in endocrinology and metabolism: TEM

Shipston MJ.
PMID: 18406709
Trends Endocrinol Metab. 1995 Oct;6(8):261-6. doi: 10.1016/1043-2760(95)00149-2.

Adrenal glucocorticoid hormones, released in response to stress activation of the hypothalamic-pituitary-adrenal axis (HPA), are powerful regulators of cellular function. Analysis of early (10 min to

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Shipston MJ. Mechanism(s) of early glucocorticoid inhibition of adrenocorticotropin secretion from anterior pituitary corticotropes. Trends Endocrinol Metab. 1995;6(8):261-6doi: 10.1016/1043-2760(95)00149-2.
Shipston, M. J. (1995). Mechanism(s) of early glucocorticoid inhibition of adrenocorticotropin secretion from anterior pituitary corticotropes. Trends in endocrinology and metabolism: TEM, 6(8), 261-6. https://doi.org/10.1016/1043-2760(95)00149-2
Shipston, M J. "Mechanism(s) of early glucocorticoid inhibition of adrenocorticotropin secretion from anterior pituitary corticotropes." Trends in endocrinology and metabolism: TEM vol. 6,8 (1995): 261-6. doi: https://doi.org/10.1016/1043-2760(95)00149-2
Shipston MJ. Mechanism(s) of early glucocorticoid inhibition of adrenocorticotropin secretion from anterior pituitary corticotropes. Trends Endocrinol Metab. 1995 Oct;6(8):261-6. doi: 10.1016/1043-2760(95)00149-2. PMID: 18406709.
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Stress, acute hyperglycemia, and hyperlipidemia role of the autonomic nervous system and cytokines.

Trends in endocrinology and metabolism: TEM

Nonogaki K, Iguchi A.
PMID: 18406806
Trends Endocrinol Metab. 1997 Jul;8(5):192-7. doi: 10.1016/s1043-2760(97)00038-6.

Stress is accompanied by metabolic alterations that could contribute to the etiology of diabetes mellitus, arteriosclerosis, and cardiovascular diseases; however, the mechanisms by which stress affects glucose and lipid metabolism remain to be resolved. Stress-induced effects on neurotransmission and...

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Nonogaki K, Iguchi A. Stress, acute hyperglycemia, and hyperlipidemia role of the autonomic nervous system and cytokines. Trends Endocrinol Metab. 1997;8(5):192-7doi: 10.1016/s1043-2760(97)00038-6.
Nonogaki, K., & Iguchi, A. (1997). Stress, acute hyperglycemia, and hyperlipidemia role of the autonomic nervous system and cytokines. Trends in endocrinology and metabolism: TEM, 8(5), 192-7. https://doi.org/10.1016/s1043-2760(97)00038-6
Nonogaki, K, and Iguchi, A. "Stress, acute hyperglycemia, and hyperlipidemia role of the autonomic nervous system and cytokines." Trends in endocrinology and metabolism: TEM vol. 8,5 (1997): 192-7. doi: https://doi.org/10.1016/s1043-2760(97)00038-6
Nonogaki K, Iguchi A. Stress, acute hyperglycemia, and hyperlipidemia role of the autonomic nervous system and cytokines. Trends Endocrinol Metab. 1997 Jul;8(5):192-7. doi: 10.1016/s1043-2760(97)00038-6. PMID: 18406806.
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Showing 1 to 12 of 707 entries